AustralieFrV1, Main, Exploration, bibRecord, 009910

Resistance and the jumping gene

Identifieur interne : 009910 ( Main/Exploration ); précédent : 009909; suivant : 009911

Resistance and the jumping gene

Auteurs : Richard Ffrench-Constant [Royaume-Uni] ; Philip Daborn [Australie] ; Rene Feyereisen [France]

Source :

BioEssays [ 0265-9247 ] ; 2006-01.

RBID : ISTEX:3FBE45B3EEF4A5366CA97B5ED69DBC85D6B16167

Descripteurs français

Wicri :
- topic : Insecticide, Pesticide.

English descriptors

KwdEn :
- Acad, Accord element, Accord insertion, Adaptive, Allele, Aminetzach, Bioessays, Bogwitz, Cause overtranscription, Cause resistance, Chkov1, Chkov1 gene, Choline, Choline acetyltransferase, Choline metabolism, Cytochrome, Daborn, Different genomic location, Drosophila, Drosophila melanogaster, Ectopic recombination, Exciting alternative, Gene, Gene product, Genetic change, Insecticide, Insecticide resistance, Insecticide targets, Insertion, Long terminal, Melanogaster, Metabolic, Metabolic enzyme, Metabolic enzymes, Molecular biology, Natl, Natural population, Natural selection, Overtranscription, Pesticide, Proc, Proc natl acad, Putative, Resistant strain, Transposable, Transposable element, Transposable elements, Transposon, Transposon insertion, Wiley periodicals.
Teeft :
- Acad, Accord element, Accord insertion, Adaptive, Allele, Aminetzach, Bioessays, Bogwitz, Cause overtranscription, Cause resistance, Chkov1, Chkov1 gene, Choline, Choline acetyltransferase, Choline metabolism, Cytochrome, Daborn, Different genomic location, Drosophila, Drosophila melanogaster, Ectopic recombination, Exciting alternative, Gene, Gene product, Genetic change, Insecticide, Insecticide resistance, Insecticide targets, Insertion, Long terminal, Melanogaster, Metabolic, Metabolic enzyme, Metabolic enzymes, Molecular biology, Natl, Natural population, Natural selection, Overtranscription, Pesticide, Proc, Proc natl acad, Putative, Resistant strain, Transposable, Transposable element, Transposable elements, Transposon, Transposon insertion, Wiley periodicals.

Abstract

Transposons are well‐known architects of genetic change but their role in insecticide resistance has, until recently, only been speculated upon.1 Transposon insertion, or transposon‐mediated transposition, could alter either metabolic enzymes capable of degrading pesticides or could change the functionality of insecticide targets. The recent work of Aminetzach and coworkers2 suggests an exciting alternative, that transposon insertion can cause resistance by altering gene product function. This hypothesis is discussed in the light of other examples in which transposons have been implicated in insecticide resistance. BioEssays 28:6–8, 2006. © 2005 Wiley Periodicals, Inc.

Url:

https://api.istex.fr/document/3FBE45B3EEF4A5366CA97B5ED69DBC85D6B16167/fulltext/pdf

DOI: 10.1002/bies.20354

Affiliations:

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Le document en format XML

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<front><div type="abstract" xml:lang="en">Transposons are well‐known architects of genetic change but their role in insecticide resistance has, until recently, only been speculated upon.1 Transposon insertion, or transposon‐mediated transposition, could alter either metabolic enzymes capable of degrading pesticides or could change the functionality of insecticide targets. The recent work of Aminetzach and coworkers2 suggests an exciting alternative, that transposon insertion can cause resistance by altering gene product function. This hypothesis is discussed in the light of other examples in which transposons have been implicated in insecticide resistance. BioEssays 28:6–8, 2006. © 2005 Wiley Periodicals, Inc.</div>
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Resistance and the jumping gene

Resistance and the jumping gene

Source :

Descripteurs français

English descriptors

Abstract

Links toward previous steps (curation, corpus...)

Le document en format XML

Pour manipuler ce document sous Unix (Dilib)

Pour mettre un lien sur cette page dans le réseau Wicri